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STMN3 Monoclonal antibody, PBS Only

STMN3 Monoclonal Antibody for WB, Indirect ELISA

Host / Isotype

Mouse / IgG2b

Reactivity

human

Applications

WB, Indirect ELISA

Conjugate

Unconjugated

CloneNo.

1F12B1

Cat no : 66152-1-PBS

Synonyms

SCG10 like protein, SCLIP, Stathmin 3, stathmin like 3, STMN3

Validation Data Gallery

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  • Western Blot (WB) analysis of human brain tissue using STMN3 Monoclonal antibody (66152-1-Ig)

    WB analysis of human brain using 66152-1-Ig (same clone as 66152-1-PBS)

    human brain tissue were subjected to SDS PAGE followed by western blot with 66152-1-Ig (STMN3 antibody) at dilution of 1:1000 incubated at room temperature for 1.5 hours. This data was developed using the same antibody clone with 66152-1-PBS in a different storage buffer formulation.

Product Information

66152-1-PBS targets STMN3 in WB, Indirect ELISA applications and shows reactivity with human samples.

Tested Reactivity human
Host / Isotype Mouse / IgG2b
Class Monoclonal
Type Antibody
Immunogen STMN3 fusion protein Ag18844
Full Name stathmin-like 3
Calculated Molecular Weight 21 kDa
Observed Molecular Weight 21 kDa
GenBank Accession NumberBC009381
Gene Symbol STMN3
Gene ID (NCBI) 50861
Conjugate Unconjugated
Form Liquid
Purification MethodProtein A purification
Storage Buffer PBS Only
Storage ConditionsStore at -80°C.

Background Information

Stathmin family phosphoproteins participate in the control of microtubule dynamics and have been proposed to be involved in the control of neuronal differentiation. Neuron specific Stathmin-3 (STMN3) is a novel STAT3 (signal transducer and activator of transcription 3) interacting protein. STAT3 is a key contributor to cancer cell migration and invasion. STMN3 exhibits microtubule-destabilizing activity, which is antagonized by STAT3. SCG10 is a widely studied and recognized neuronal differentiation marker, STMN3 is a SCG10-like protein (SCLIP) and is present from the earliest stages of hippocampal neuron differentiation in culture at vesicle-like structures following dynamic microtubules. RNAi mediated inhibition of this gene resulted in increased axonal branching.